Professor and Chair
Office: 336-334-5313
email: d_kipp@uncg.edu
Ph.D., Cornell University
R.D., Miami Valley Hospital, Dayton, OH
The role of nutrients, bioactive food components, and hormones in regulating osteoblast function and bone remodeling, using animal models and cell culture.
- Age-related prolactin effects on the skeleton.
- Effects of iron imbalance on bone development.
- Effects of bioactive food components on bone formation.
- Messer, J.G., Cooney, P.T., Kipp, D.E. Iron chelator deferoxamine alters iron-regulatory genes and proteins and suppresses osteoblast phenotype in fetal rat calvaria cells. Bone 2010;46(5):1408-15).
- Messer, J.G., Kilbarger, A.K., Erikson, K.M., Kipp, D.E. Iron overload alters iron-regulatory genes and proteins, down-regulates osteoblastic phenotype, and is associated with apoptosis in fetal rat calvaria cultures. Bone 2009;45(5):972-9.
- Kipp, D.E., Grey, C.E., McElvain, M.E., Kimmel, D.B., Robinson, R.G., and Lukert, B.P. Long-term low ascorbic acid intake reduces bone mass in guinea pigs. J. Nutr. 1996;126:2044-2049.
- Kipp, D.E., McElvain, M., Kimmel, D.B., Akhter, M.P., Robinson, R.G. and Lukert, B.P. Scurvy results in decreased collagen synthesis and bone density in the guinea pig animal model. Bone 1996;18:281-8.
- Pate, S.K., Lukert, B.P. and Kipp, D.E. Tissue vitamin C levels of guinea pig offspring are influenced by maternal vitamin C intake during pregnancy. J. Nutr. Biochem.1996;7:524-8.